Difference between revisions of "Publications"
From PastDB
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__NOTOC__ | __NOTOC__ | ||
− | ==== | + | ==== PastDB publication ==== |
+ | * Martín, G., Marquez, Y., Duque, P., Irimia, M. (2020). Alternative splicing landscapes in Arabidopsis thaliana across tissues and stress conditions highlight major functional differences with animals. Submitted. | ||
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+ | ==== VastDB publication (v1.0) ==== | ||
* Tapial, J., Ha, K. C. H., Sterne-Weiler, T., Gohr, A., Braunschweig, U., Hermoso-Pulido, A., Quesnel-Vallières, M., Permanyer, J., Sodaei, R., Marquez, Y., Cozzuto, L., Wang, X., Gómez-Velázquez, M., Rayon, T., Manzanares, M., Ponomarenko, J., Blencowe, B. J., Irimia, M. (2017). An atlas of alternative splicing profiles and functional associations reveals new regulatory programs and genes that simultaneously express multiple major isoforms. ''Genome Research, 27''(10), 1759–1768. [[http://genome.cshlp.org/content/27/10/1759.long link]] [[https://www.ncbi.nlm.nih.gov/pubmed/28855263 PubMed]] | * Tapial, J., Ha, K. C. H., Sterne-Weiler, T., Gohr, A., Braunschweig, U., Hermoso-Pulido, A., Quesnel-Vallières, M., Permanyer, J., Sodaei, R., Marquez, Y., Cozzuto, L., Wang, X., Gómez-Velázquez, M., Rayon, T., Manzanares, M., Ponomarenko, J., Blencowe, B. J., Irimia, M. (2017). An atlas of alternative splicing profiles and functional associations reveals new regulatory programs and genes that simultaneously express multiple major isoforms. ''Genome Research, 27''(10), 1759–1768. [[http://genome.cshlp.org/content/27/10/1759.long link]] [[https://www.ncbi.nlm.nih.gov/pubmed/28855263 PubMed]] | ||
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==== Intron retention analysis ==== | ==== Intron retention analysis ==== | ||
* Braunschweig, U., Barbosa-Morais, N.L., Pan, Q., Nachman, E., Alipahani, B., Gonatopoulos-Pournatzis, T., Frey, B., Irimia, M., Blencowe, B.J. (2014). Widespread intron retention in mammals functionally tunes transcriptomes. ''Genome Research'', 24:1774-86. [[http://genome.cshlp.org/content/early/2014/09/24/gr.177790.114 link]] [[http://www.ncbi.nlm.nih.gov/pubmed/25258385 PubMed]] | * Braunschweig, U., Barbosa-Morais, N.L., Pan, Q., Nachman, E., Alipahani, B., Gonatopoulos-Pournatzis, T., Frey, B., Irimia, M., Blencowe, B.J. (2014). Widespread intron retention in mammals functionally tunes transcriptomes. ''Genome Research'', 24:1774-86. [[http://genome.cshlp.org/content/early/2014/09/24/gr.177790.114 link]] [[http://www.ncbi.nlm.nih.gov/pubmed/25258385 PubMed]] | ||
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Latest revision as of 15:57, 2 July 2020
PastDB publication
- Martín, G., Marquez, Y., Duque, P., Irimia, M. (2020). Alternative splicing landscapes in Arabidopsis thaliana across tissues and stress conditions highlight major functional differences with animals. Submitted.
VastDB publication (v1.0)
- Tapial, J., Ha, K. C. H., Sterne-Weiler, T., Gohr, A., Braunschweig, U., Hermoso-Pulido, A., Quesnel-Vallières, M., Permanyer, J., Sodaei, R., Marquez, Y., Cozzuto, L., Wang, X., Gómez-Velázquez, M., Rayon, T., Manzanares, M., Ponomarenko, J., Blencowe, B. J., Irimia, M. (2017). An atlas of alternative splicing profiles and functional associations reveals new regulatory programs and genes that simultaneously express multiple major isoforms. Genome Research, 27(10), 1759–1768. [link] [PubMed]
vast-tools
- Tapial, J., Ha, K. C. H., Sterne-Weiler, T., Gohr, A., Braunschweig, U., Hermoso-Pulido, A., Quesnel-Vallières, M., Permanyer, J., Sodaei, R., Marquez, Y., Cozzuto, L., Wang, X., Gómez-Velázquez, M., Rayon, T., Manzanares, M., Ponomarenko, J., Blencowe, B. J., Irimia, M. (2017). An atlas of alternative splicing profiles and functional associations reveals new regulatory programs and genes that simultaneously express multiple major isoforms. Genome Research, 27(10), 1759–1768. [link] [PubMed]
- Irimia, M., Weatheritt, R.J., Ellis, J., Parikshak, N.N., Gonatopoulos-Pournatzis, T., Babor, M., Quesnel-Vallières, M., Tapial, J., Raj, B., O’Hanlon, D., Barrios-Rodiles, M., Sternberg, M.J.E., Cordes, S.P., Roth, F.P., Wrana, J.L., Geschwind, D.H., Blencowe, B.J. (2014). A highly conserved program of neuronal microexons is misregulated in autistic brains. Cell, 59:1511-23. [link] [PubMed]